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MEDICAL REHABILITATION MANAGEMENT OF CARPAL TUNNEL SYNDROME Imam Subadi; Hanik Hidayati; Fidiana Fidiana; Nur Sulastri
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Carpal tunnel syndrome (CTS), the most common entrapment neuropathy in the upper extrimity, is a clinical syndrome characterized by a tingling sensation, numbness, pain, or weakness in the hand and wrist radiating up to the arm. This condition is a major cause of absenteeism, reduced productivity, and financial loss among various neuropathy due to median nerve compression. This paper, medical rehabilitation of CTS is viewed. Medical rehabilitation of CTS aimed to reduce pain, reduce clamping of the carpal tunnel, sensory and motor reeducation so that hand function improves and can perform activities of daily living. Treatment options can be given includes exercise therapy, ultrasound diathermy, low level laser therapy (LLLT), and shock wave therapy (SWT).

CENTRAL VERTIGO Sela Pricilia; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Central vertigo is a symptom characterized by a feeling of changes in body position or environment as a result of diseases originating from the central nervous system. Central vertigo is caused by a disease that extend from vestibular nuclei in medulla oblongata to ocular motor nuclei and integration system in mesencephalon to vestibulocerebellum, thalamus and vestibular cortex in temporoparietal and the neuronal pathway which mediate VOR (vestibulo-ocular reflex). The diseases can be vestibular migrain, TIA (Transient Ischemic Attack), Vertebrobasilar ischemic stroke, multiple sclerosis, tumor in cerebelopontine angle and congenital malformation like Dandy Walker Syndrome. Central vertigo can be diagnosed by performing several special tests. This examination can also distinguish central vertigo from its differential diagnosis, namely peripheral vertigo. Management of central vertigo can be in the form of acute attack management and specific management according to the cause.

COMPARISON OF HYDRODISECTION INJECTION BETWEEN TRIAMCINOLONE ACETONIDE VERSUS DEXAMETHASONE IN CARPAL TUNNEL SYNDROME Widodo Mardi Santoso; Badrul Munir; Catur Ari Setianto; Ria Damayanti; Sheny Agma
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Background: Carpal tunnel syndrome (CTS) is the most common nontraumatic peripheral neuropathy, which is caused by suppression of the median nerve below the transverse carpi ligament. Local corticosteroid injection is considered the fastest and most effective method for improving symptoms that occur in CTS. There are several corticosteroid agents that can be used, but there are no objective standards that can explain the most ideal drugs. Objective: To compare the effectiveness of hydrodisection injection therapy of triamcinolone acetonide versus dexamethasone on carpal tunnel syndrome. Methods: This study involved 30 participants who were diagnosed with CTS and fulfilled the inclusion criteria and no exclusion criteria were obtained. Participants were divided into two treatment groups; the first group (n = 15) injected with Triamcinolone Acetonide (TCA) 10mg / 1ml and lidocaine 1% 1 ml and the second group (n = 15) injected with Dexamethasone 4mg / 0.8ml and lidocaine 1% 1 ml. The NRS, FSS, and SSS parameters were assessed before injection and 4 weeks after injection in each agent. Then compared these parameters at 4 weeks after injection compared to the TCA group with the dexamethasone group. Results: NRS score before and 4 weeks after TCA injection (sig 0.000; p <0.05), SSS (sig 0.001; p <0.05) and FSS (sig 0.020; p <0.05), and NRS score before and 4 weeks after dexamethasone injection (sig 0.001; p <0.05), SSS (sig 0,000; p <0.05) and FSS (sig 0,000; p <0.05). At 4 weeks after injection of TCA compared to dexamethasone there were no significant results on NRS (sig 0.237; p> 0.05) and FSS (sig 0.119; p> 0.05), while SSS values were significantly different (sig 0.027; p <0.05). Conclusion: Significant improvement in NRS, FSS and SSS score was obtained at 4 weeks after hydrodisection injection, both with TCA and dexamethasone. At 4 weeks after TCA injection compared to dexamethasone, there were no significant differences in NRS and FSS scores, whereas SSS score differed significantly. Both injection agents are equally effective in treating CTS, but dexamethasone produces a better improvement in SSS score.

COMPLICATED MIGRAINE Kadek Putri Paramita Abyuda; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Migraine is a chronic paroxysmal neurological disease characterized by attacks of moderate or severe headache accompanied by reversible neurologic and systemic symptoms. Although not life threatening, migraine can cause disability in the productive population. Migraine sufferers generally have a family history of migraine so that migraine is considered a genetic disease. Endogenous psychological factors such as stress or fatigue are the main triggers for migraine. Migraine pathophysiology involves various parts of the brain so that migraine symptoms are complex. Management of acute migraine can be done pharmacologically and non-pharmacologically. Migraine preventive management is needed if the patient has a chronic migraine or does not respond to abortive treatment.

VESTIBULAR NEURONITIS Shahdevi Nandar Kurniawan; Afiyfah Kaysa Waafi
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Vestibular neuronitis is an acute vestibular syndrome due to inflammation of the vestibular nerve characterized by the typical symptoms of acute rotatory vertigo accompanied by nausea, vomiting, and symptoms of balance disorders. The incidence of vestibular neuronitis is about 3.5 per 100,000 people. The exact etiology of this vestibular neuronitis is unknown. However, based on existing evidence, vestibular neuronitis is associated with viral infections of the upper respiratory tract and herpes zoster infection. The clinical manifestations of vestibular neuronitis are persistent rotatory vertigo accompanied by oscillopsia, horizontal-rotatory peripheral vestibular spontaneous nystagmus on the healthy side, and a tendency to fall on the affected side. Diagnosis of vestibular neuronitis can be made by clinical diagnosis, through history, physical examination, and special examinations. Through these examinations, the differential diagnosis of vestibular neuronitis should be excluded, such as Meniere's disease, labyrinthitis, benign paroxysmal positional vertigo, and vertigo due to central lesions such as cerebellar infarction. Management of vestibular neuronitis is in the form of symptomatic therapy with vestibular suppressants, antivertigo, and redirect to relieve the symptoms that arise, then causative therapy can be done by administering corticosteroids, and in patients, physiotherapy can be done to improve vestibular function.

MEDICAL REHABILITATION MANAGEMENT OF CARPAL TUNNEL SYNDROME Imam Subadi; Hanik Hidayati; Fidiana Fidiana; Nur Sulastri
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.3

Carpal tunnel syndrome (CTS), the most common entrapment neuropathy in the upper extrimity, is a clinical syndrome characterized by a tingling sensation, numbness, pain, or weakness in the hand and wrist radiating up to the arm. This condition is a major cause of absenteeism, reduced productivity, and financial loss among various neuropathy due to median nerve compression. This paper, medical rehabilitation of CTS is viewed. Medical rehabilitation of CTS aimed to reduce pain, reduce clamping of the carpal tunnel, sensory and motor reeducation so that hand function improves and can perform activities of daily living. Treatment options can be given includes exercise therapy, ultrasound diathermy, low level laser therapy (LLLT), and shock wave therapy (SWT).

CENTRAL VERTIGO Sela Pricilia; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.4

Central vertigo is a symptom characterized by a feeling of changes in body position or environment as a result of diseases originating from the central nervous system. Central vertigo is caused by a disease that extend from vestibular nuclei in medulla oblongata to ocular motor nuclei and integration system in mesencephalon to vestibulocerebellum, thalamus and vestibular cortex in temporoparietal and the neuronal pathway which mediate VOR (vestibulo-ocular reflex). The diseases can be vestibular migrain, TIA (Transient Ischemic Attack), Vertebrobasilar ischemic stroke, multiple sclerosis, tumor in cerebelopontine angle and congenital malformation like Dandy Walker Syndrome. Central vertigo can be diagnosed by performing several special tests. This examination can also distinguish central vertigo from its differential diagnosis, namely peripheral vertigo. Management of central vertigo can be in the form of acute attack management and specific management according to the cause.

COMPARISON OF HYDRODISECTION INJECTION BETWEEN TRIAMCINOLONE ACETONIDE VERSUS DEXAMETHASONE IN CARPAL TUNNEL SYNDROME Widodo Mardi Santoso; Badrul Munir; Catur Ari Setianto; Ria Damayanti; Sheny Agma
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.1

Background: Carpal tunnel syndrome (CTS) is the most common nontraumatic peripheral neuropathy, which is caused by suppression of the median nerve below the transverse carpi ligament. Local corticosteroid injection is considered the fastest and most effective method for improving symptoms that occur in CTS. There are several corticosteroid agents that can be used, but there are no objective standards that can explain the most ideal drugs. Objective: To compare the effectiveness of hydrodisection injection therapy of triamcinolone acetonide versus dexamethasone on carpal tunnel syndrome. Methods: This study involved 30 participants who were diagnosed with CTS and fulfilled the inclusion criteria and no exclusion criteria were obtained. Participants were divided into two treatment groups; the first group (n = 15) injected with Triamcinolone Acetonide (TCA) 10mg / 1ml and lidocaine 1% 1 ml and the second group (n = 15) injected with Dexamethasone 4mg / 0.8ml and lidocaine 1% 1 ml. The NRS, FSS, and SSS parameters were assessed before injection and 4 weeks after injection in each agent. Then compared these parameters at 4 weeks after injection compared to the TCA group with the dexamethasone group. Results: NRS score before and 4 weeks after TCA injection (sig 0.000; p <0.05), SSS (sig 0.001; p <0.05) and FSS (sig 0.020; p <0.05), and NRS score before and 4 weeks after dexamethasone injection (sig 0.001; p <0.05), SSS (sig 0,000; p <0.05) and FSS (sig 0,000; p <0.05). At 4 weeks after injection of TCA compared to dexamethasone there were no significant results on NRS (sig 0.237; p> 0.05) and FSS (sig 0.119; p> 0.05), while SSS values were significantly different (sig 0.027; p <0.05). Conclusion: Significant improvement in NRS, FSS and SSS score was obtained at 4 weeks after hydrodisection injection, both with TCA and dexamethasone. At 4 weeks after TCA injection compared to dexamethasone, there were no significant differences in NRS and FSS scores, whereas SSS score differed significantly. Both injection agents are equally effective in treating CTS, but dexamethasone produces a better improvement in SSS score.

COMPLICATED MIGRAINE Kadek Putri Paramita Abyuda; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.2

Migraine is a chronic paroxysmal neurological disease characterized by attacks of moderate or severe headache accompanied by reversible neurologic and systemic symptoms. Although not life threatening, migraine can cause disability in the productive population. Migraine sufferers generally have a family history of migraine so that migraine is considered a genetic disease. Endogenous psychological factors such as stress or fatigue are the main triggers for migraine. Migraine pathophysiology involves various parts of the brain so that migraine symptoms are complex. Management of acute migraine can be done pharmacologically and non-pharmacologically. Migraine preventive management is needed if the patient has a chronic migraine or does not respond to abortive treatment.

VESTIBULAR NEURONITIS Shahdevi Nandar Kurniawan; Afiyfah Kaysa Waafi
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.5

Vestibular neuronitis is an acute vestibular syndrome due to inflammation of the vestibular nerve characterized by the typical symptoms of acute rotatory vertigo accompanied by nausea, vomiting, and symptoms of balance disorders. The incidence of vestibular neuronitis is about 3.5 per 100,000 people. The exact etiology of this vestibular neuronitis is unknown. However, based on existing evidence, vestibular neuronitis is associated with viral infections of the upper respiratory tract and herpes zoster infection. The clinical manifestations of vestibular neuronitis are persistent rotatory vertigo accompanied by oscillopsia, horizontal-rotatory peripheral vestibular spontaneous nystagmus on the healthy side, and a tendency to fall on the affected side. Diagnosis of vestibular neuronitis can be made by clinical diagnosis, through history, physical examination, and special examinations. Through these examinations, the differential diagnosis of vestibular neuronitis should be excluded, such as Meniere's disease, labyrinthitis, benign paroxysmal positional vertigo, and vertigo due to central lesions such as cerebellar infarction. Management of vestibular neuronitis is in the form of symptomatic therapy with vestibular suppressants, antivertigo, and redirect to relieve the symptoms that arise, then causative therapy can be done by administering corticosteroids, and in patients, physiotherapy can be done to improve vestibular function.

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Contact Name
Shahdevi Nandar Kurniawan

Contact Email
shahdevinandar@ub.ac.id

Phone
+62341-321297

Journal Mail Official
jphv@ub.ac.id

Editorial Address
Neurology Department, Faculty of Medicine, Brawijaya University Jl. JA Suprapto No. 2 Malang, Indonesia 65112

Location
Kota malang,

Jawa timur

INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name Shahdevi Nandar Kurniawan

Contact Email shahdevinandar@ub.ac.id

Phone +62341-321297

Journal Mail Official jphv@ub.ac.id

Editorial Address Neurology Department, Faculty of Medicine, Brawijaya University Jl. JA Suprapto No. 2 Malang, Indonesia 65112

Location Kota malang, Jawa timur INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Contact Name
Shahdevi Nandar Kurniawan

Contact Email
shahdevinandar@ub.ac.id

Phone
+62341-321297

Journal Mail Official
jphv@ub.ac.id

Editorial Address
Neurology Department, Faculty of Medicine, Brawijaya University Jl. JA Suprapto No. 2 Malang, Indonesia 65112

Location
Kota malang,

Jawa timur

INDONESIA